Ингибирование гиперпродукции оксида азота в условиях прогрессивно нарастающей гипоксии на фоне действия ИЛ-1β снижает выживаемость крыс после острой гипоксии

The role of the nitric oxide (NO) in the compensatory reactions the organism to increasing acute normobaric hypoxia was evaluated in anesthetized Wistar rats with an increased proinflammatory cytokine interleukin-1beta (IL-1β) levels. A progressively increasing hypoxia from normoxia up to apnea was ... View More