Mitochondrial uncoupling does not decrease reactive oxygen species production after ischemia-reperfusion.
Published in American journal of physiology. Heart and circulatory physiology • Oct 1, 2014
NobleIDNI7P20W32R10S87
Authors:,,
Ricardo Quarrie
Daniel S Lee
Levy Reyes
Abstract
Cardiac ischemia-reperfusion (IR) leads to myocardial dysfunction by increasing production of reactive oxygen species (ROS). Mitochondrial H(+) leak decreases ROS formation; it has been postulated that increasing H(+) leak may be a mechanism of decreasing ROS production after IR. Ischemic preconditi...
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